

On
the diagram at the head of this page, Type I periodontal disease is
characterized by swollen and red gums. In fact, this condition
is called gingivitis. The distance between
the crest of the gingiva and the depth of the sulcus is greater than that found in healthy gums, but this is caused
mostly by swelling of the gingival tissues. Increased sulcus depth
caused by swelling of the gingiva, rather than by loss of bone is called pseudopocketing. If there is a substantial buildup
of calculus in the sulcus, the treatment of choice is a full
mouth debridement followed by a second prophylaxis visit (referred
to as a fine scale). A debridement procedure (covered above) accomplishes two goals:
It allows for initial healing of the pseudopockets so that the dentist/hygienist
can do an accurate pocket charting at the next visit, and it allows
the patient to clean to the base of the sulcus using ordinary toothbrush
and floss. In most cases, serious gingivitis responds well to
this regimen and is permanently cured if the patient practices good
daily dental hygiene.
Swelling subsides and the depth of the pocket returns to normal.
There is not yet any destruction of bone, and thus not much root is
exposed, so the debridement procedure can usually be done without local
anesthesia.
In
type II periodontal disease, some bone loss has occurred. The
sulcus depth is now increased due to both swollen gingiva and the loss
of the bone. Whenever the depth of the sulcus is increased due
to the loss of bone, the term "sulcus" is replaced with the
term "pocket". In early periodontal
disease, the pockets tend to be localized to the areas between the teeth
and are at most 3-4 mm deep. As with type I disease, the first
line of treatment always involves a debridement visit followed by the
thorough removal of all calculus and plaque from the root surfaces in
several succeeding visits. Because of the bone loss, however,
more root is exposed and local anesthesia may be needed. This
type of scaling is called a root planing. Once
again, with good continued hygiene, the pocket depth will subside due
to the reduction in swelling of the gingiva.
The
difference between early periodontal disease and moderate periodontal
disease is the increased depth and distribution of the pocketing.
In moderate periodontitis, pocket depths (or attachment loss) are 4
to 6 mm. There is generally bleeding upon probing, and sometimes
slight tooth mobility.
These cases are also treated with an initial debridement visit followed by several visits for scaling and root planing procedures. Unfortunately, because of the loss of the bone, the pockets may not subside all the way back to normal. Floss may not be able to reach all the way down to the base of the pocket between the teeth (where most of the problem generally starts). The key to cure is good hygiene, and that means cleaning the plaque all the way to the bottom of the pocket (see next paragraph). At this point, the disease may be kept permanently at bay by taking special measures to be sure to clean to the depth of the pocket around each and every tooth.
These measures include the use of the rubber tips frequently found on the ends of the handle of toothbrushes, Stimudents, Doctor's BrushPics and Proxabrushes. Water Picks can be useful, but they are not as effective as mechanical devices like good old fashioned toothpicks. "Sonic" electric toothbrushes are fairly effective at cleaning deeply on the outside and inside surfaces, but they will not reach completely between the teeth. If good hygiene is practiced all the time, the pocket depths will continue to decrease over time, and patients who have had type III periodontal disease will, sooner or later, find themselves with teeth showing more exposed root due to the gingival crest receding because of increasing gingival health, but with no active disease processes continuing to destroy the periodontal tissues. If the disease continues due to some remaining deep pocketing which prevents complete cleaning with good home care, the patient is often referred to a specialist for surgery to eliminate the pockets. These patients are now most often treated regularly with adjunctive antibiotic therapy and Arestin placed in the deeper pockets.
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| Teeth & Bone in health | Type IV periodontal disease |
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Type
IV periodontal disease is much more serious than either of the other
two types because the bone loss is so much more pronounced. It
involves not only gingival (soft tissue) pocketing, but also "infrabony"
pocketing which is diagramed in the type III schematic
at the head of this page, and can be seen quite well on the image of
the dentaform to the right. The roots of many of the teeth are
sitting in "wells" of bone. Consequently, much of the
bone that surrounds the roots does not actually touch them and therefore
lends no support. Even patients suffering type IV periodontal
disease are initially treated with an initial debridement visit followed
by several visits (generally 4 visits, one for each quadrant) of root
planing.
Even in severe cases like this, a thorough root planing followed by excellent oral hygiene can generally stop the progression of the disease. The major difficulty here is that the bony pockets will not rebuild, and it becomes very difficult to reach all the way to the bottom of the infrabony pockets to clean them. In most of these cases, patients are referred to a periodontist who is a "gum specialist" who will perform periodontal surgery to modify the shape of the gums so that good oral hygiene can be accomplished. These patients are are also treated regularly with adjunctive antibiotic therapy and Arestin placed in the deeper pockets.
Patients in this category may start out with mild to moderate periodontitis, follow through with treatment for each stage, carry out all home care procedures and get routine periodontal scaling every three or four months, but still suffer progressive bone loss into and through type IV periodontitis. We say they are refractory because the normal periodontal treatments may slow down the progression of the disease, but do not halt it, even with the best home and professional care.
Recent studies have found that this type of patient may have a genetic propensity toward periodontal disease, as well as coronary artery disease and cerebrovascular disease. Approximately 35% of all people possesses the genetic interlukin-1 marker. The IL-1 marker is a key regulator of the inflammatory process.
These people are best treated with a combination of regular periodontal root planings, periodontal surgery and adjunctive antibiotic therapy targeting the specific bacterial species which are initiating the inflammatory response. This is covered in more detail below.